AR-NTD

Royal Marsden and Johann de Bono

Advancing AR-NTD disease biology with clinical prostate cancer leadership at The Royal Marsden and ICR.

Illustrative generated image of a Victorian red-brick hospital facade with gold lettering reading The Royal Marsden Hospital, representing the Chelsea campus context. Not a documentary photograph of the building.
Institution
The Royal Marsden and The Institute of Cancer Research
Location
London, United Kingdom
Principal investigator
Prof. Johann de Bono
Regius Professor of Cancer Research
Program
AR-NTD
Focus
  • Castration-resistant prostate cancer
  • AR splice variants
  • Biomarker-driven translation

Why we partner

AR-NTD binders must be understood in the same resistance landscape that defines modern prostate cancer care. We partner with Prof. Johann de Bono at The Royal Marsden and The Institute of Cancer Research to connect Peptone's disordered-domain chemistry to clinical disease biology, including the splice-variant and biomarker contexts that decide whether a next-generation androgen receptor therapy can matter for patients.

Scientific focus

Prof. de Bono leads prostate cancer targeted therapy and drug development programs that have shaped the treatment of advanced disease. His group's work on androgen receptor aberrations, including constitutively active splice variants such as AR-V7, established why therapies that only occupy the ligand-binding domain leave a critical escape route open. That clinical and molecular framing is essential for AR-NTD mechanism-of-action studies.

What Peptone brings

Peptone brings small molecules designed against the intrinsically disordered N-terminal domain, characterised by ensemble measurement rather than a static pocket assumption. The platform supplies compounds, biophysical engagement data, and a clear hypothesis: shut down the transcriptional activation function that resistance mechanisms continue to exploit.

What we do together

The collaboration focuses on mechanism of action and disease-relevant biology: how Peptone AR-NTD compounds behave in models and experimental systems informed by clinical resistance, circulating biomarkers, and the genomic landscape of lethal prostate cancer. The aim is to pressure-test MoA early, with access to translational insight that is difficult to recreate in isolation.

Therapeutic ambition

We collaborate to build first-in-class AR-NTD therapeutics with a clearer path from measured binding to patient-relevant biology. Access to cutting-edge clinical prostate cancer models and biomarker thinking ahead of the field is the point of the partnership: deepen disease understanding so the right molecules advance, and the wrong ones do not.